Hello everyone,

I have been doing alot of studies in chimera, mainly by following  
individual cell types using lacZ reporters.  One of the defects I see  
is very strange - ecmA:lacZ (construct S) in the ecmO collar region.   
Even more strangely is that in a mutant/wild type ecmA:lacZ mix, it is  
the wild type cells that go there!

Has anyone ever seen anything like it with things they are working on  
or know of any papers where a similar phenotype has been seen?  Its a  
difficult search term and i'm not having much luck.

Cheers
Alex